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|| Early Breast Development || Menstrual Irregularity || Infertility || PC OD || Excess Menstrual Bleeding ||
 || Premature Ovarian Failure || Premenstrual Syndrome || Menopause || Hormone Replacement Therapy ||


Menstrual Irregularity

Introduction & Normal Physiology of Menstruation

Absent Periods (Amenorroea)
Late Periods (Oligomenorroea)

Introduction: Menses irregularity may be complete absence of menstruation which is called amenorrhea or there may be delayed menstruation which is called oligomenorrhoea.

Normal Physiology of Menstruation: In normal female there are lacs of immature egg (ovarian) follicle lying in dormant state in the ovary. During the normal menstrual cycle on third day of cycle approximately 30 egg follicles starts maturing finally one of them will mature to full stage to form mature egg. When all the hormones are normal then one out of all maturing egg follicle is selected for further maturation & rest all shrink & atrophy. Finally this selected egg follicle passes through various stages of development & finally matures into a completely developed egg called ovum. Then egg is released out of the ovary (i.e. ovulation). Once the ovum is released ovary starts secreting progesterone for next 10 days. This progesterone hormone leads to strengthening of walls of blood vessel of endometrium. After 10 days of egg release i.e. ovulation, the production of progesterone hormone from ovary ceases. Thus the sudden deficiency of progesterone hormone leads to weakening of the linings of endometrial blood vessels which ultimately shrink & stop supplying blood to endometrium i.e. inner uterine lining. Thus in nut shell the production of estrogen from ovary lads to formation of inner lining of the uterus during menstrual cycle, where as production of progesterone from ovary after the ovulation leads to maintenance of this uterine lining. The menstrual bleeding occurs due to shedding of uterine inner lining i.e. endometrium due to disruption of its blood supply because of sudden deficiency of progesterone because of stoppage of its production from ovary. Thus if no ovulation occurs, there will not be any cyclical exposure of progesterone resulting in absence of regular menstrual bleeding. 
For detail information about causes, investigation & treatment click following links:


Amenorrhoea (means absence of period)
Late Periods (Oligomenorroea)


Amenorrhoea 

Amenorrhoea term is used when there is total absence of menses. 
Absence of period can occur due to any of the following hormone disorder as estradiol, pituitary hormone LH, FSH, Prolactin & Thyroid hormone, T3, T4, TSH etc due to any of the below mentioned causes. 

Causes of Amenorrhoea are:


1.Ovarian failure (hypergonadotropic hypogonadism): Gonadal agenesis, Gonadal dysgenesis, Turner syndrome 45, X, Mosaicism, Pure gonadal dysgenesis, 46, XX, 46,XX (Swyer syndrome), Ovarian enzymatic deficiency as 17a-Hydroxylase deficiency, 17, 20-Lyase deficiency, Premature Ovarian failure : Idiopathic – premature menopause, injury, mumps oophoritis, radiation, chemotherapy, Resistant ovary (Savage syndrome), Autoimmune disease, Galactosemia. 

2. Absence of ovulation: Polycystic ovarian syndrome (PCOS or PCOD),Hyperthecosis, Adrenal disease : Cushing syndrome, adult-onset adrenal hyperplasia, Thyroid disease : Hypothyroidism, hyperthyroidism, Ovarian tumors : Granulosa-theca cell tumors, Brenner tumors, cystic teratomas, mucinous/serous cystadenomas, Krukenberg tumors. 

3. Hypothalmic Pituitary Abnormality leading to absence of estrogen production (hypogonadotropic hypogonadism). Craniopharyngioma, Germinoma, Hamartoma, Hand Schuller Christan disease, Teratoma, Endodermal, sinus tumors, metastatic carcinoma, Infection and other disorders : Tuberculosis, Syphilis, Encephalitis/meningitis, sarcoidosis, kallmann syndrome, idiopathic hypogonadotropic hypogonadism, Chronic debilitating disease, 
Pituitary Other hormone-screening pituitary tumors (ACTH, thyrotropin-stimulating hormone, growth hormone), non-functional tumors (craniophyaryngioma), Metastatic carinoma, Space-occupying lesions : Empty sella syndrome, Arterial aneuysm, Necrosis : Sheehan syndrome, Panhypopituitarism, Inflammatory /infiltrative : Sarcoidosis, Hemachoromatosis. 

4. Prolactinomas & Hyperprolactinemia

5. Defect in uterus or vagina: Labial agglutination/fusion, Imperforate hymen, transverse vaginal septum, Cervical agenesis – isolated, Cervical stenosis – iatrogenic, vaginal agenesis – isolated, Mullerian agenesis (Mayer-Rokitansky-Kuster-Hauser syndrome), Complete androgen resistance testicular feminization), Endrometrial hypoplasia or aplasia – congenital, Asherman syndrome (uterine synechiae), small hypoplastic uterus etc. 

Investigations & Diagnosis:


For diagnosis of cause of absent menses we do following tests.
Complete hormone profile: LH, FSH, total testosterone, free testosterone, Prolactin, Androstenidione, SHBG, 17 HYDROXYPROGESTERONE, DHEAS, Estradiol, Progesterone
Suppression & Stimulation Tests, 
Karyotype
Genetic testing
Pro. Challenge test 
Hystero-salpingography for uterus & tube status 
Laproscopy
High Resolution Ultrasound

Treatment:-Treatment of specific diseases. Once the cause for Amenorrhoea is found then treatment started. Abnormalities of the thyroid gland, pituitary and abnormalities in the adrenal glands can be treated by appropriate medicines. Normally patient starts getting normal menstrual cycle (period) in three to six month time. The various drugs which are required are as follows: 
a) Clomiphene
b) Gonadotropin Therapy
c) HCG Therapy
d) Hmg therapy
e) Metformin
f) Glitazones
g) Tamoxifene
h) Letrozole
i) Medroxyprogesterone
i) Desgestrol
j) Megestrol
k) Norethisterone
l) Hydroxyprogesterone
m) Oral E & P Combination Pill

Late Periods (Oligomenorroea)

Late periods are called oligomenorrea & this is a common problem during reproductive years in female. Menstrual irregularity can occur due to any of the following hormone disorder as estradiol, pituitary hormone LH, FSH, Prolactin & Thyroid hormone, T3, T4, TSH etc due to any of the below mentioned causes.

What are causes of menstrual irregularity?

- Abnormal function of the hypothalamus. Decreased secretion of Gonadotropin results in menstrual irregularity ; This problem can occur due to Craniopharyngioma, Germinoma, Hamartoma, Hand Schuller Christan disease, Teratoma, Endodermal, sinus tumors, metastatic carcinoma, Infection and other disorders : Tuberculosis, Syphilis, Encephalitis/meningitis, sarcoidosis, kallmann syndrome, idiopathic hypogonadotropic hypogonadism, Chronic debilitating disease, 

- Abnormal functions of the pituitary gland results in abnormal secretion of some hormones such as prolactin, LH, FSH leading ro high prolactin or low FH, FSH. Other hormone-screening pituitary tumors (ACTH, thyrotropin-stimulating hormone, growth hormone), non-functional tumors (craniophyaryngioma), Metastatic carinoma, Space-occupying lesions : Empty sella syndrome, Arterial aneuysm, Necrosis : Sheehan syndrome, Panhypopituitarism, Inflammatory /infiltrative : Sarcoidosis, Hemachoromatosis. 

- High values of prolactin hormone called hyperprolactinemia due to prolactinoma, idiopathic hyperprolactinemia or due to other causes is an important cause of disturbed periods.

- Abnormal functions of the ovaries: Decreased function of the ovaries results in increased blood level of follicle Stimulating Hormone, Failure of ovaries before the age of 35 years is called premature ovarian failure. Aproximately 5% women have stoppage of period by 35 years. Causes of disturbed ovarian function are Polycystic ovarian syndrome (PCOS or PCOD),Hyperthecosis, Adrenal disease : Cushing syndrome, adult-onset adrenal hyperplasia, Thyroid disease : Hypothyroidism, hyperthyroidism, Ovarian tumors : Granulosa-theca cell tumors, Brenner tumors, cystic teratomas, mucinous/serous cystadenomas, Krukenberg tumors. , Gonadal dysgenesis, Turner syndrome 45, X, Mosaicism, Pure gonadal dysgenesis, 46, XX, 46,XX (Swyer syndrome), Ovarian enzymatic deficiency as 17a-Hydroxylase deficiency, 17, 20-Lyase deficiency, Premature Ovarian failure : Idiopathic – premature menopause, injury, mumps oophoritis, radiation, chemotherapy, Resistant ovary (Savage syndrome), Autoimmune disease, Galactosemia. 

Immaturity of hypothalmic pituitary ovarian system 

What are the laboratory tests for menstrual irregularity?
Recommend some of the following laboratory tests confirm the diagnosis :
- Hormone Tests: L.H., F.S.H., Prolactin, Estradiol (E2),
Increase in Male hormone as Testosterone, Free testosterone, DHEAS, Androstenidione, 17.0 H.P. SHBG
- Thyroid function tests, Excess or deficient functions of thyroid glands can either cause decreased menstruation or amenorrhea.
Ultrasonography of the pelvis region for ovarian size & uterus.
Some other tests may also be required depending on history & physical examination

What is the treatment for menstrual irregularity?
- Treatment of specific diseases. Abnormalities of the thyroid gland, pituitary and abnormalities in the adrenal glands can be treated by appropriate medicines.
- Progesterone withdrawal

- Hormone therapy. Hormone treatment is recommended for that particular hormone disorder which is detected during hormone investigation. The various hormone therapies are 
a) Clomiphene
b) Gonadotropin Therapy
c) HCG Therapy
d) Hmg therapy
e) Metformin
f) Glitazones
g) Tamoxifene
h) Letrozole
i) Medroxyprogesterone
i) Desgestrol
j) Megestrol
k) Norethisterone
l) Hydroxyprogesterone
m) Oral E & P Combination Pill
n) Metformin
o) Glitazones

For consultation & treatment (by appointment / or online) click How to Consult Us

                                                                                            

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At these places all the facilities for investigation & treatments are available.

At our center / centre / clinic / clinics / hospital / hospitals doctors are dedicated for latest & scientific treatment of these diseases. 

Dexamethasone Suppression test for ACTH, Overnight Suppression (Basal at 8-9am on day oral 1mg dexa given at night 11pm. Post dexa sample coiiectea at 8-9am next day.), L ow dose (Basal at 8-9am on day 1. Oral 0.5mg dexa given, every 6 hrs for 2 days. Post dexa sample collected at 8-9am on day-3, High dose (Basal at 8-9am on day 1. Oral 2 mg dexa given every 6 hrs for 2 days. Post dexa sample collected at 8-9am on day-3, Dexamethasone Suppression test for Cortisol: - Overnight Suppression (Basal at 8-9am on day 1. Oral 1mg, dexa given at night 11pm. Post dexa sample collected at 8-9am next day.), Low dose (Basal at 8-9am on day 1. Oral O.5mg dexa given every 6 hrs. for 2 days. Post dexa sample collected at 8-9am on day-3, High dose (Basal at 8-9am on day 1. Oral 2 mg dexa given every 6hours for 2 days. Post dexa sample collected at 8-9am on next day.), DHEAS-Dihydroepiandrosteindione Sulphate, serum, DHT-Di hydro testosterone, serum, Diabetes profile, Mini (FBS, PPBS, Cholesterol, HbA1c, Microalbumin), Diabetes profile.Maxi (CBC, FBS, PPBS, Cholesterol, Triglycerides, Creatinine, Electrolytes, Urine it, Insulin antibody, Microalbumin, Insulin, C-peptide, HbA1c), Dialysis panel (CBC, FBS, Electrolytes, Proteins, Alkaline phosphatase, Ca, P, Uric acid, Creatinine, BUN), Diazepam (Benzadiazepine), urine (Qualitative), Digoxin (Lanoxin), serum, Dilantin (Phenyntoin) (Eptoin), serum, DNA (Double Strand) Antibody, serum (Qualitative), Down’s Syndrome Screen-1 st trimester (Triple test) (Down’s syndrome screening through analysis of Free HCB-beta, PAPPa and statistical risk factor calculation), Down’s Syndrome Screen-2nd trimester (Triple test) (Down’s syndrome screening through analysis of HCB-beta, AFP, uE3 and statistical risk factor calculation), DPD-Deoxypyridinoline, urine, Drugs of abuse panel (Qualftative)(7 Drug panel) (Amphetamine, Coccaine, Barbiturates, Cannabinoids, Opiates, PCP and Benzodiazaplne), Drugs of abuse panel (QuantitativeXS Drug panel) (Amphetamine, Methamphetamine, Coccaine, Barbiturates, Cannabinoids, Opiates, PCP, Benzodiazapine and Methadone with creatinine correction), DVT-Deep Vein Thrombosis Panel (Activity of AntiThrombin-III, Protein C and Protein S, Lupus anticoagulant, Cardiolipin IgG and IgM), D-xylose test, E2 - Estradiol, serum, E3-Unconjugated Estriol, serum, EBV(VCA)-lgM antibodies, Echinococcus (Hydatid Cyst) - IgG, serum, ECG, Echovirus antibody-IgG, Echovirus antibody-IgM, EGF-Epidermal Growth Factor, Electrolytes, ENA(Extractable Nuclear Antigen) profile (Antibodies to SSA, SSB, Sm, RNP-Sm, Scl-70, Jo-1), Endocrine panel-Female (FSH, LH, Prolactin, E2 and TSH to be done on 2-5th day of cycle.)., Endocrine panel-Male (FSH, LH, Prolactin, Testosterone-total & free and TSH), Endothelial Markers Profile (Factor VIII, CD31, CD34), Endomysial Antibody, Epinephrine(Adrenaline), Epinephrine (Adrenaline),Epithelial Marker profile(Pan Keratin,CK10,EMA,CEA), , ithelial Membrane Ag(EMA)-lmmunohistochemistry, toin (Phenytoin). serum, ER-Estrogen receptor,  Erythropoietin, serum (EPO), ESR (Automated), Estradiol (E2), serum, Ethanol, Quantitative (Analyse same day), FABP-Fatty acid binding protein (Cardiac risk marker), Factor V leiden (Activity), plasma,  Factor V leiden, mutant detection,  Factor VIII (v/W factor), Factor VIII activity, Factor-13, plasma, Faeces, Routine examination, Fc gamma R2 receptor, Fc gamma R3a receptor,  FDP-Fibrinogen Degradation Products Febrile agglutination test,  Ferritin, serum, Fever profile (Maxi) (CBC, Urine rt, ESR, MP, SGPT, Widal, ___ Aerobic blood culture), Fever profile (Mini) (CBC, Urine rt, ESR, MP, Widal),  Fibrinogen, plasma, Fibro test, Fibro test and Acti test, Filaria antigen (Wuchereria Bancrofti), FISH for AML (M2, M3 and M4) {Includes t(15;17), t(8;21) and inv(16)}, FISH for del 13q (For multiple myeloma), FISH for Her2/Neu (Prognostic marker for CA breast), FISH for inv(16) (For Acute Myeloid Leukaemia AML M4), FISH for t ( 8;21 )(For Acute Myeloid Leukamia AML M2), FISH for t ( 9 ; 22 ) (For bcr-abl translocation of Philadelphia Chromosome), FISH for t (15 ; 17 ) (For promyelocytic leukaemia (PML/RARA/AML M3), File Stain for histopathology, FNAC - cytological examination, FNAC procedure charges (For walk in patients), Foetal Haemoglobin (HbF), blood, Folic acid (RBC, Serum and whole blood), Folic acid, Serum, Food analysis, bacteriological, Free androgen index (FAI), Free Beta HCG, Serum, Free PSA (Prostate Specific Antigen-Free molecule), Free Testosterone, Serum, Frozen Section (At Lab) for histopathology, Frozen section (Intra operative) for histopathology, Frozen section (Intra operative) for histopathology, Fructose (Qualitative), semen, FSH- Follicle Stimulating Hormone, Serum,

FSH-LH-Prolactin-serum, FSH-LH-Prolactin-Testosterone, serum, FSH-LH-Testosterone, serum, FTA-Abs (IgG antibodies to Treponema), Serum, Fungal culture with identification, G6PD-Quantitative, blood, Gamma GT (GGTP), Serum, Cardinal (Pnenobarbitone) , serum by CLIA, Gastric Parietal Cell Antibody, serum, Gastrin, serum (Fasting), Gastrin, serum (Post prandial), GBM - Antibody to Glomerular Basement Membrane, Gentamicin (Garamycin/Genticyn/Lyramycin), German Measles(Rubella), Avidity test, serum, German Measles(Rubella), IgG antibody, German Measles(Rubella), IgM antibody, Gl mucosal biopsy (SOS special stain), Giardia antigen Stool, Giemsa Stain for histopathology, Gilal Fibrillary Acidic Protein, Gliadin - IgA antibodies, Gliadin - IgG antibodies, Glucagon, Glucagon Stimulation test for C-peptide test, Glucose, Glycogen Phosphorylase (Cardiac risk marker), Glycoprotein C, Glycated Haemoglobin (HbA1c), blood, GnRH Stimulation test (Basal and 30-60-90-120 minutes sample after iv GnRH injection for FSH & LH tests) (Dose = 2.5uo/Ka of body weight), Gram’s staining, Growth Hormone, Serum (Fasting required), GTC-Glucose Tolerance Curve (5 samples), GTC-Glucose Tolerance Curve, Extended (8 samples) , Glucose Challenge Test, GTT for Pregnancy (100 gm Glucose) (Four Samples), GTT of 5 samples (75 gm. of glucose), Haemoglobin Electrophoresis, blood, Haemoglobin, blood, Haemoglobin, free, Haemogram, blood,  lHanm’s test (Acidified serum test), Hanging drop for V. cholerae,  Haptoglobulin, serum, HAV-IgG Antibodies to Hepatitis A virus, Serum,  HAV-IgM Antibodies to Hepatitis A virus, Serum, HbA1C-Glycated haemoglobin, blood, HbcAb-IgM antibodies to  Hepatitis B Core Antigen

HbcAb-Total antibodies to  Hepatitis B Core Antigen

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Osmania General Hospital, Hyderabad. Gandhi Hospital, Secunderabad. Government Maternity Hospital, Hyderabad. Nilofar Hospital, Hyderabad. Cancer Hospital, Hyderabad. T.B. Hospital, Irrumnuma, (Andhra Pradesh). Hospital for Mental Diseases, Hyderabad. Sarojini Devi Hospital, Hyderabad. Government General Hospital, Guntur. Government General Hospital, Kurnool. Government General Hospital, Kakinada. M.G.M. Hospital, Warangal. Govt. Hospital, Tirupati.Government Hedquarters Hospital, Anantapur, (Andhra Pradesh). Government Headquarters Hospital, Elur, (Andhra Pradesh). K.G. Hospital, Vishkhapatnam. T.B. Hospital, Vishkhapatnam. Mantal Hospital, Vishkhapatnam. Victoria Hospital for Women & Children, Vizag, (Andhra Pradesh). Cantonment Hospital, Bolarum (Andhra Pradesh). Railway Hospital, Vijayawada. S.V. Ramnarain Ruia Hospital, Tirupati. S.V. Ram Narain Ruia Govt. General Hospital, Tirupati. Government Women & Children Hospital, Tirupati. Railway Hospital, Secunderabad. Government Headquarters Hospital, Rajahmundry, (Andhra Pradesh). Dist. Headquarters Hospital, Nizamabad, (Andhra Pradesh). Headquarters Hospital, Nellore, (Andhra Pradesh). Govt. Hospital, Vijayawada. Govt. Headquarter Hospital, Machilipatnam, (Andhra Pradesh). Medwin Hospital, Hyderabad.

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(continued) thyroid function parameters , treatment hCG , IL-2 , thymosin Alpha1 wasting syndrome , GH resistance , recombinant human growth hormone , sex differences , testosterone therapy , muscle mass, thalidomide , treatment , HIV protease inhibitor, lipodystrophy caused by, HL. Human leukocyte antigen(s) Hodge, Gary D., Hodgkin disease testicular failure, treatment gonadal toxicity , thyroid dysfunction caused by, type 1 Gaucher disease , HOE 140, HOE 901, Hobauer cells, Hollander syndrome, Hollander syndrome, Holmes, Edward W., Holmes-Adie syndrome, Holocrine secretion, Holoprosencephaly,. Septo-optic dysplasia Holotelencephaly, Holst, Jens J., Home blood glucose monitoring, devices , disadvantages , frequency testing , glycemic control, pregnancy, Homoestasis, definition , homeostasis [term], Homocarnosinase, barin, deficiency , homocarnosinosis, Homocysteine metabolism , plasma/serum lefel , hypothyroidism, retinal vascular occlusive disease, reference values , Homocystinuria, childhood ostopenia due , classic, clinical findings , clinacl mangemet , cutaneous manifestations , definition , diagnosis , eyelids , genetic , with hypomethininemia, infancy childhood, metabolic defect , ocular manifestations , osteoporasis , secondary cobalamin metabolism defects, organ-specific involvement , skeletal abnormalities , thromboembolic disease, urine screening test , homogentisis acid oxidase deficiency, cutaneous manifestations , ocular effects o, Homologous recombination, Homonymous hermianopia, with pituitary adenoma, Homoplasmy, Homosexua male(s) hypothalamic structure , urogenital infection , Homovanillic acid (HVA) reference values , structure , synthesis ,synthesis , urinary children, infants children, age-related changes , Honeymoon period, insulin therapy, HONK. Hyperosmolar hyperglycemic nonketotic coma Hoover, Robert N., Hormokine, Hormnal deficiency syndrome(s), hypoglycemia , Hormonal mediator(s), mast cell-dependent. Mediator(s) Hormonal obesity, hormone(s), actions , calcium ad, modulation or lateration , phasphatidylinositde metabolites , receptor-mediated, recepteor or postreceptor mediation , pathologic physiologic effects , amino-acid derivatives as, biossays , central nervous system effects , ciculating, menstrual cycle, classification , as cocarcinogens, cutaneous biotransformation , deciduas, definition , diversity , generation , endogenous, allergy , premenstrual syndrome, exogenous, ocular effects , expression , norma, abnormal, fatty acid derivatives as, free, assays , group I, features , mechanism action , group II, features , mechanis action , groups IIA, group IIB, group IIC, historical perspective , hypothalamic, hypopituitarism, inactivation , pathologic physiologic effects , lipophilic, maternal levels , pregnancy, measurement , factors affecting, mechanism action , measage , modulation or alteration , momenclature , peptide. peptide hormone(s) placental, polypeptide, puberty, femal, pulmonary, female, pulmonary, disease, functions , lung cancer, peripheral blood, reproductive, menopause , role , salivary levels , secretion , pulsatile, study , methods , regulation , sources , steroid, synthesis , by immune system cells, regulation , therapy with, infections, transport , alterations , pathologic physiologic effects , water-soluble Hormone [term], hormone receptor(s),. specific hormone cell surface, coupling domain , desensitization , down-regulation , feedback systems, multisystem control proximity endocrine glands , functional domains , immune system cells, intracellular, loss , occupancy , bioeffect, phosphorylation , recognition domain , regulation , selectivity , sepecificity , structure , subtypes , transport proteins, comparison , up-regulatio , hormone receptor-G-protein effector system, subunits , Hormone-regulatory elements (HREs), Hormone replacement therapy (HRT). Estrogen replacement therapy bleeding with, bone density, breast cancer, cardioprotective effects , corticosteroid-induced osteoporosis, fracture risk reduction, lipid levels, menopausal, endometrial cancer risk, osteogenesis imperfecta, osteoporosis, monitoring , biomarkers bone turnover , ovarian cancer, plus parathyroid hormone, bone mass, postmenopausal, cardiovascular effects , postmenopausal women, lipid profile , procollagen type I propeptide levels , rheumatoid arthritis, systemic lupus erythematosus, vaginal blood flow, XY/XO gonadal dysgenesis hormone resistance, Gs deficiency , hormone response element (HREs), Hormone response units (HRUs), Horner syndrome, causes , pituitary tumor , pregnancy, Hortobagy, Gabriel N., Horvath, Eva, Hospitalization, catecholamine levels, Hot flashes/ hot flushes clomiphene , menopause,


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