Small Breast & Treatment 

What is small breast?
Causes of small breast
How normal breast Develops
Investigations & Diagnosis
Treatment Options for Breast Enlargement
Hormone & Medicine Treatments
Breast Enlargement Surgeries
Response of treatment

Causes of Small Breast: In girls the small breast can be due to following causes:

1) Female genital hormones deficiency i.e. estradiol in body due to many of the below mentioned causes as leading to absence of estrogen production (hypogonadotropic hypogonadism). Craniopharyngioma, Germinoma, Hamartoma, Hand Schuller Christan disease, Teratoma, Endodermal, sinus tumors, metastatic carcinoma, Infection and other disorders : Tuberculosis, Syphilis, Encephalitis/meningitis, sarcoidosis, kallmann syndrome, idiopathic hypogonadotropic hypogonadism, Chronic debilitating disease, Pituitary other hormone-screening pituitary tumors (ACTH, thyrotropin-stimulating hormone, growth hormone), non-functional tumors (craniophyaryngioma), Metastatic carinoma, Space-occupying lesions : Empty sella syndrome, Arterial aneuysm, Necrosis : Sheehan syndrome, Panhypopituitarism, Inflammatory /infiltrative : Sarcoidosis, Hemachoromatosis. Gonadal agenesis, Gonadal dysgenesis, Turner syndrome 45, X, Mosaicism, Pure gonadal dysgenesis, 46, XX, 46,XX (Swyer syndrome), Ovarian enzymatic deficiency as 17a-Hydroxylase deficiency, 17, 20-Lyase deficiency, Premature Ovarian failure : Idiopathic – premature menopause, injury, mumps oophoritis, radiation, chemotherapy, Resistant ovary (Savage syndrome), Autoimmune disease, Galactosemia. 

2) Second cause for small breast is resistance of breast tissue to normal level of female hormone in the body. So that even though female hormone is normal in the body, but the breast tissue of these girls do not have sufficient receptors for female hormone leading to insufficient estrogen action meaning that female hormone is unable to work & breast do not grow well. But when female hormone level is further increased by female hormone replacement treatment & certain more medicines are given so that female hormones becomes more effective on breast, then breast tissue starts growing. Breast size then fully normalizes in six months to one-year time. 
3) Growth factor deficiency
4) Zinc Deficiency
5) Estrogen biosynthetic defects
6) Congenital adrenal hyperplasia
7) Congenital absence of breast bud
8) Lipoatrophy generalized
9) Ninth Cause is resistance of hair roots to normal level of male hormones, in this disorder male hormone is normal in blood even then hair root do not grow because in hair root there is less male hormone receptor (i.e. sensor of male hormone) so that male hormone is unable to work & hair do not grow well. But when male hormone level is further increased by male hormone replacement treatment & certain more medicines are given so that male hormones becomes more effective on hair roots, then hair roots starts growing. Beard & moustaches then fully normalizes in six months to one-year time. 
10) Absence of estrogen receptors in breast tissue
12) Growth hormone deficiency due to hypopituiarism, pituitary disorders
13) Progeria
12) DRUGS: Cytotoxic Agents, Cyclophoamide, Vincristine,Ionising radiation, Azathioprine
13) Overmastectomy 
14) Structural Defects Of body fat cell so that they do not grow
15) Generalized hypoplastic syndrome
16) Dysmorphic Syndrome

Investigations & Diagnosis:

 For diagnosis of cause of absent or small breast we do following tests.

Complete hormone profile: LH, FSH, total testosterone, Prolactin, Androstenidione, SHBG, 17 HYDROXYPROGESTERONE, DHEAS, Estradiol, Progesterone

 Suppression & Stimulation Tests,Karyotype
Genetic testing
Choromosome analysis
Pro. Challenge test
High Resolution Ultrasound
Mammography
E2 CHALLENGE TEST
Diagnostic Therapeutic Response Test

Treatment Options for Breast Enlargement

Before starting treatment for enlargement of breast all above causes are investigated and proper diagnosis is made. Then we test whether breast enlargement is possible or not.

First We Test Whether Breast Enlargement Is Possible Or Not 

After above investigation we make the decision about whether breast enlargement possible or not. We prescribe the hormone treatment only if the above investigations suggest for the scope of further breast enlargement.

Diagnostic Therapeutic Response Test

This procedure is required in some patient, if above test still does not clarifies of the possibility of breast enlargement. For this hormone & other treatment is given for three month in the form of female hormone tablet, local hormone application at breast skiin, or vaginal gels & additional other therapy. If breast enlarges in size i.e. it grows in size by more than 2 cm in these three months, it means breast of that patient has potential for further growth. Once it is decided by above test that breast has potential for further growth, in such patient further courses of hormone therapy is given. By few courses of hormone treatment breast enlarges to normal size in approximately one year time. By this therapy breast size as well its consistency in firmness also increases. Thus breast becomes large in size, more attractive & develops firm protruding look. This breast enlargement treatment is very effective in those patient’s in whom investigations & physical examination shows significant abnormality. Thus most patients achieve fully normal breast. As breast size increases in size patient's confidence increases and feeling of well-being is achieved.

How this treatment works:-When we give below-mentioned hormones by any of the below-mentioned routes. The concentration of these hormones in blood circulation rises. This leads to binding of more hormones on estrogen receptors of breast. After binding with receptors these hormones leads to certain changes in the breast tissue. This leads to multiplication of breast tissue leading to further growth. This continued multiplication of breast tissue leads to further breast growth. This continued multiplication of breast tissue under the effect of above mentioned hormone treatment continues till breast size normalizes. Usually in one-year time breast size as well as texture i.e. firmness normalizes. Unluckily most doctors have less knowledge about hormone treatment for this problem & also they think the problem of small breast as more of cosmetic problem so that they neither investigate the patient nor give hormone or other medical therapy for breast growth.

Various therapeutic options of breast enlargement are available as:

This treatment can be either:
a. Hormone & Other Medicine Therapy: available as pills, hormonal skiin patches or vaginal creams & other adjuvant drugs are prescribed
OR
b. Surgical therapy: Breast enlargement surgeries
Hormone & Medicine Treatment of Small Breast 
We start medical treatment only when above tests shows that breast has capacity to enlarge. It is given in the form replacement of female hormones & other hormone as growth hormone & growth factor replacement. These hormones can be given in the form of oral tablets, skiin patches, ointments, gel or creams. 
As this treatment has no side effects, is economical and easy to take either as oral tablets, skiin patches, or as gel, thus it is most preferred form of breast enlargement therapy.

The various Treatment options are: 
Treatment of specific disease: Once the cause for absent or small breast is found by above tests then treatment started. Abnormalities of the pituitary and abnormalities in the adrenal glands can be treated by appropriate medicines. Normally patient starts getting improvement in breast size in three to six months.
The various drugs which are required are as follows: 

1) Female hormones replacement: The replacement of female hormones is one of the common treatments for breast enlargement. The various female hormones are as follows: 
a) Estradiol
b) Conjugated equine estrogen
c) Estriol
d) Natural estrogens
e) Prohormones
f) Estrogen skiin patches
g) Estrogen vaginal creams
h) Medroxyprogesterone
i) Oral E & P Combination Pill
j) Desgestro
k) Megestrol
L) Norethisterone
N) Hydroxyprogesterone

2) Growths Hormones Therapy: Growth hormone is given when somatotropin deficiency is found & that was cause of small breast..
3) IGF-1 therapy: is given when somatomedin-c deficiency is the cause. 
4) Gonadotropin therapy is also very effective in breast enlargement in many patient.
5) Gonadotropin releasing hormone therapy is effective in many cases of under developed breast.
6) Medicine to increase the fat in breast tissue & body.

But unluckily many doctors do not take the problem seriously leading to patient not given proper treatment. Thus many patients remain having small size of breast & associated lifelong inferiority complex. 

Response of Medical Treatment: 

In properly selected patient, when any of above treatment is started the response is very good & has no side effects. In many scientific studies when the above treatment started breast size increases from previous size of hardly visible breast to as big as 36 inches & in some even larger size of 38 or 40 inches. Thus the trial of treatment should be given to all patients with small breast before asking them to go for surgery. It has no side effects if given in proper disease and in-patients when it is really indicated. 

Breast Enlargement Surgery 

Surgical Treatment is required when medicine are not effective then there are various surgical procedures which can lead long lasting good size firm breast. Our Cosmetic surgeon performs all breast increasing surgery.

Female Hormone Deficiency

Female Hormone Deficiency (Hypogonadism) is called when female hormone estradiol is less in body. The symptoms of female hormone deficiency are: menstrual irregularity, small breast, decreased desire for genital, dryness of vagina or pain during intercourse.

Cause of female hormone deficiency are hypothalamic disease, pituitary failure, L.H., F.S.H deficiency, prolactin excess, functional, hypothalamic amenorrhoea, defective egg formation or no egg formation at all or ovarian failure due to many of the causes. (hypogonadotropic hypogonadism). Craniopharyngioma, Germinoma, Hamartoma, Hand Schuller Christan disease, Teratoma, Endodermal, sinus tumors, metastatic carcinoma, Infection and other disorders : Tuberculosis, Syphilis, Encephalitis/meningitis, sarcoidosis, kallmann syndrome, idiopathic hypogonadotropic hypogonadism, Chronic debilitating disease, Pituitary other hormone-screening pituitary tumors (ACTH, thyrotropin-stimulating hormone, growth hormone), non-functional tumors (craniophyaryngioma), Metastatic carinoma, Space-occupying lesions : Empty sella syndrome, Arterial aneuysm, Necrosis : Sheehan syndrome, Panhypopituitarism, Inflammatory /infiltrative : Sarcoidosis, Hemachoromatosis. Gonadal agenesis, Gonadal dysgenesis, Turner syndrome 45, X, Mosaicism, Pure gonadal dysgenesis, 46, XX, 46,XX (Swyer syndrome), Ovarian enzymatic deficiency as 17a-Hydroxylase deficiency, 17, 20-Lyase deficiency, Premature Ovarian failure : Idiopathic – premature menopause, injury, mumps oophoritis, radiation, chemotherapy, Resistant ovary (Savage syndrome), Autoimmune disease, Galactosemia.
 
Investigation: For diagnosis of cause of absent or small breast we do following tests.
Complete hormone profile: LH, FSH, total testosterone, Prolactin, Androstenidione, SHBG, 17 HYDROXYPROGESTERONE, DHEAS, Estradiol, Progesterone

Suppression & Stimulation Tests, 
Karyotype
Genetic testing
Choromosome analysis
Pro. Challenge test 
High Resolution Ultrasound
Mammography
E2 CHALLENGE TEST
Diagnostic Therapeutic Response Test

Treatment: After finding out the cause various hormone defects are corrected. Some short cause of female hormone is given. After treatment of approximately six months body starts forming hormone itself in many cases. Breast development starts uterus starts growing. In next three one year complete development of female genital character develops & menstrual period starts. 

1) Female hormones replacement: The replacement of female hormones is one of the common treatments for breast enlargement. The various female hormones are as follows: 
a) Estradiol
b) Conjugated equine estrogen
c) Estriol
d) Natural estrogens
e) Prohormones
f) Estrogen skiin patches
g) Estrogen vaginal creams
h) Medroxyprogesterone
a) Desgestrol
b) Megestrol
c) Norethisterone
d) Hydroxyprogesterone
e) Oral E & P Combination Pill
2) Gonadotropin therapy is also very effective in breast enlargement in many patient.

3) Gonadotropin releasing hormone therapy is effective in many cases of under developed breast.

Response of Treatment: After treatment of approximately six months body starts forming hormone itself in many cases. Breast development starts uterus starts growing. In next three one year complete development of female genital character develops & menstrual period starts.
 

Breast development at early age (Precocious Puberty) 

Precocious Puberty :Breast development alone or additional genital development may occur at early age early age (termed as Precocious Puberty). 
Causes: 
If puberty occurs early then there is risk that her height may remain short & some psychological problem may occur. Sometimes threr may be risk of genital exploitation by others as child is still innocent even with good genital development.

After hormone testing the particular hormone defect is corrected leading to disappearance of breast & pubic hair. Later breast development & menstrual period comes at right time. So In all such cases one should try to find out exact cause of hormone disorder & then treatment should be started there are many suppressant hormone i.e. GnRH analogues which when given to girls then breast & of pubic hair regresses back.

Causes of Precocious Puberty

This can be due to increase in female hormone at very young age due to: 
Ovarian cause, or due to pituitary-hypothalmic disorder as hypothalmic harmartoma, ovarian tumors, congenital adrenal hyperplasia & adrenal tumour & other CNS disorder. It can be due to idiopathic central precocious puberty (i.e. true) or pseudo precocious puberty. Other causes are premature thelarche, & premature pubarche.

Central (GnRH) driven : Idiopathic (sporadic or familial), Central nervous system abnormalities,Acquired (Abscess, chemotherapy, granulomas, hypothalamic hamartomas, sept-optic dysplasia, suprasellar cyst.Tumor (LH-secreting adenoma, astrocytoma, slioma (may be associated with neurofibromatosis), crainpharyngiomas, ependymomas. 
Secondary to chronic exposure to genital steroid (causes of peripheral puberty, CVAH, GIP, tumors) Reversible forms : space-occupying or pressure-associated lesions (Abscess, hydrocephalus) Peripheral (GnRH independent) : Genetic disorders (mutations) , Congenial virlizing adrenal hyperplasia (CVAH), males, Gonadotropin-independent puberty (GIP), males, McCune-Albright syndrome. 
Tumors : Adrenal genital steroid secreting (adenoma, carcinoma), gonadotropin-producing (choriocarcinoma, chorioepithelioma, dysgerminoma, hepatoblastoma, hepatoma, teratoma) Ovarian (granulose cell, may be associated with Peutz-Jeghers syndrome); granulose, theca cell Testicular (Leydig Cell) 

Limited or reversible forms : Chronic, primary hypothyroidism, CVAH, Exogenous genital steroid or gonadotropins, Ovarian cysts.

Variants of normal development : Premature pubarche (Secondary to premature adrenarche), Premature thelarche.

Investigations & Diagnosis:

For diagnosis of cause of absent or small breast we do following tests.
Complete hormone profile: LH, FSH, total testosterone, Prolactin, Androstenidione, SHBG, 17 HYDROXYPROGESTERONE, DHEAS, Estradiol, Progesterone
Suppression & Stimulation Tests, 
GnRH Stimulation test
Pro. Challenge test 
High Resolution Ultrasound
Mammography
Diagnostic Therapeutic Response Test
MRI

Treatment Options for Early Puberty 

Treatment of specific disease: Once the cause for early genital development is found by above tests then treatment started. Abnormalities of the pituitary and abnormalities in the adrenal glands can be treated by appropriate medicines. Normally patient starts getting decrease in breast size in three to six months.
The various drugs which are required are as follows: 

1) Female hormones suppressant: GnRH analogues as triptorelin’ ,naferelin, Gonadorelin, testolactone, MEdroxy progesterone
Some times surgery may also be required

Response of treatment: Normally in three month breast size starts decreasing including pubic hair regresses. This treatment may be continued till the child puberty needs to be deferred. The moment we stop the treatment normal pubertal & genital development resumes.


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 At these places all the facilities for investigation & treatments are available.

At our center / centre / clinic / clinics / hospital / hospitals doctors are dedicated for latest & scientific treatment of these diseases. 
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AFB-Acid fast bacilli, Susceptibility-1 st line [SIREP] Includes Streptomycin, Isoniazid, Rifampicin, Etharrjbutol and Pyrizinamide)

AFB-Acid fast bacilli, Susceptibility-2nd line (Includes Kanamycin, Ethionamide, Ofloxacin, Capreomycin)

AFB-Acid fast bacilli, susceptibility: 13 drugs panel

AFB-Acid fast bacilli, Culture (U medium)

AFP-Alpha Feto Protein

Albert’s Stain for C.diphtheriae

Albert’s Stain for C.diphtheriae

Albumin, Albumin, urine (Qualitative), Albumin/Creatinine ratio, Alcian blue stain, Alcohol, Quantitative (Analyse same day), Aldehyde Cbopra test for Kala Azar, Aldolase, Aldosterone, Alkaline phosphatase with bone fraction, serum, Alkaline phosphatase, serum

Alpha 1 anti chymotrypsin for Immimohistochemistry, Alpha 2 macroglobulin, serum, Alpha HBDH, Alpha-1 -antitrypsin , serum, ALT (SGPT), serum, Aluminium, Amikacin (Amicin/Amikanex/Mikacin/Amikin), serum, Amino acids (Qualitative), Amino leveulinic acid(ALA), Urine, Amiodarone, serum, Ammonia, plasma, Amoebiasis Antibodies-IgG, serum, Amphetamine, urine (Qualitative), Amphetamine, urine (Quantitative), Amylase, ANA - Anti Nuclear Antibody, serum by EIA, ANA-Anti Nuclear Antibody, serum by IFA, ANA profile (Antibody to SSA, SSB, Sm, Jo1, UlsnRNP, Centromere, RNP-Sm and Scl-70), Anaemia Profile, Haemolytic type (CBC, Reticulocyte count, G6PD, Osmotic fragility, Haptoglobulin, Abnormal Hb studies, Coomb’s direct. ), Anaemia profile, Maxi (CBC, Reticulocyte count, lron,TIBC,Transferrin Saturation, Vitamin B12, CRP, Abnormal Hb studies, Folate(Serum&RBC), Ferritin, G6PD, Haptoglobulin, Osmotic fragility, Coomb’s-direct.), Anaemia profile, Mini (CBC, Iron, TIBC, Transferrin saturation, CRP and Ferritin)

Different Hospitals in india are: Moolchand Health Care, St. Stephen’s Hospital, Dr. Ram Manohar Lohia Hospital, G.B Pant Hospital, Lok Nayak Hospital, RG Stone Urological Reasarch Institue, Sir Ganga Ram Hospital, Jeevan Hospital, Deen Dayal Upadhyaya Hospital, Orthonova Hospital, Batra Hospital, Sahi Hospital, Indraprastha Apollo Hospitals, Holy Faimly Hospital, Jeevan Mala Hospital, Jaipur Golden Hospital, Lady Harding Hospital, Smt. Sucheta Kriplani Hospital, Mata Chanan Devi Hospital, Safdarjung Hospital, Mohindra Hospital, M.P Heart centre Hospital, Bara Hospital, Hindu Rao Hospital, Holy Angels Hospital, Shankar Hospital, Holy Family Hospital, Chopra Hospital, Escorts Hospital St. Stephen's Hospital, Vardaan Hospital, Visitech Eye Hospital, Vardhman Mahavir Medical College, Safdarjung Hospital, Lok Nayak Jai Prakash (LNJP) Hospital, Private Hospital, Guru Teg Bahadur Hospital, Kalra Hospital,  Bangalore Baptist Hospital, Bowring Hospital, B W Lions Eye Hospital, Bangalore Hospital, Bangalore Children's Hospital, Genesis Hospital, Lakeside Hospital, M G A Hospital, Mallya Hospital, Manipal Hospital, Sindhi Charitable Hospital, Yellamma Dasappa Hospital,  Sir Harkisandas Narottamdas Hospital, Prince Alikhan Hospital, Saifee Hospital, Breach Candy Hospital, Jaslok Hospital, Habib Ismail Hosptal, Kalajyot Hospital, Motiben Dalvi Hospital, Bombay Hospital, Corwest Jain Clinic Group of Hospitals, D.S. Kothari Hospital, Lotus Hospital, Elizabeth Hospital, Bhatia Hospital, The Bomanji Dinshaw Petit Parsee Hospital, Cumbala Hill Hospital, Noor Hospital, K.B. Haji Bachoali Charitable Ophthalmic & ENT Hospital, Bai Jerbai Wadia Hspital, Nawroji Wadia Maternity Hospital, Wadia Hospital, Yerla Medical Trust Hospital, Tarachand Bappa Hospital, Dhanwantari Hospital, Shri Samasta Parajiya Suvarna Hospital, Smt.B.C.J. General Hospital, Smt.B.C.J. General Hospital, Holi Spirit Hospital, Lilavati Hospital, The Bandra Holy Family Hospital, Raheja Hospital, Gurunanak Memorial Hospital, Ramakrishna Mission Hospital, Radhibai Watumull Global Hospital, BSES Hospital, Sushrut Hospital, LH Hiranandani Hospital, Maganlal Popatlal & Sarvoday Hospital, Rajawadi Municipal General Hospital, Shatabdi Hospital, KBB Hospital, V.N. Hospital, Dr.K.B. Babha Hospital, Cooper Hospital, Bhagwati Hospital, Sion Hospital, K.E.M. Hospital Nayar Hospital, Kama Hospital, G.T. Hospital, J.J. Hospital, Sr.George Hospital, Aliganj Hospital, Blue Cross Hospital, Getwell Hospital, Krishna Lok Hospital, Lucknow Hospital, Medwin Hospital and Health Centre, Vivekanand Hospital, G.M.Associate Hospital, Dufferin Hospital, Civil Hospital, Communicable/Infectious Diseases Hospital Lifeline Hospital and Heart Centre, Lucknow Hospital, Shalimar Hospital, Rajendranagar Hospital Tuberculosis Hospital, Lucknow Rajpoot Hospital, Nishat Hospital, Z A Chartable Hospital, Indira Gandhi Eye Hospital, Sitapur Eye Hospital, Awadh Hospital and Heart Centre, Balrampur Hospital, Cantonment General Hospital, Civil Hospital, Dr.Shyama Prasad Mukherjie Hospital, Fatima Hospital, G.M.Associate Hospital, K.K.Hospital, Veerangana Jhalkari Bai Female Hospital, Shekhar Hospital, Queen Mary Hospital, Osmania General Hospital, Gandhi Hospital, Government Maternity Hospital, Nilofar Hospital, Cancer Hospital, T.B. Hospital, Hospital for Mental Diseases, Sarojini Devi Hospital, Sundram Medical Foundation, Chennai. MIOT Hospital, Chennai. Apollo Hospital, Madras. Malar Hospital Limited, Gandhi Nagar Adyar, Chenni. Calcutta Medical College Hospital. N.R.S. Medical College & Hospital, Calcutta. R.G. Kar Medical College & Hospital, Calcutta. Calcutta National Medical College & Hospital, Instt. of Postgraduate Med. Education & Research, Calcutta Seth S.K.M. Hospital, Calcutta. Sambhu Nath Pandit Hospital, Calcutta. Lady Dufferinvictoria Hospital, Calcutta. Howrah General Hospital, Howrah, Hoogly Sadar Hospital, Hoogly, Chittaranjan Seva Sadan & Chittaranjan Cancer Hospital,  Ramakrishna Mission Seva Pratishthan General Hospital, Calcutta.

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carcinoma; Adrenal neoplasm(s) ultrastructure , stress, unilateral enlargement , infectious etiology, venous drainage , weight , adrenal hyperplasia. Congenital adrenal hvperplasia; 
micronodular pigirented, surgery , postoperative follow-up , primary, imaging , nodular, autonomous, MEN 1, primary pigmented, paraneoplastic syndromes, primary, primary hvperaldosteronism, stress, Adrenal hypoplasia congenita, Adrenaline. Epinephrine Adrenal insufficiency, Adrenocortical insufficiency;Adreonocorticotropic hormone (corticotropin, ACTH), deficiency ; Hypocidienalism; Hypoparathvroiclism, adrenal insufficiency, mucocutaneous candidiasis (HAM) acute clinical presentation , diagnosis , risk factors , signs symptoms , treatment , anemia , cardiovascular effects , chronic diagnosis , signs symptoms , critical illness, cutaneous manifestations , diagnosis , differential diagnosis , etiology , gastrointestinal effects ,hypothyroidism, hvpovolenlia caused bv, IL-2-related, versus .ippropriiilc intidilircsis, infections , laboratory findings , mitotane-ind need, neuromuscular menifestaitions , otolarynggologix consideration , pernicious anemia , postpartuin, pregnancy, primary, Addison disease adrenal morphology , definition , eitilogy hypothyroidism, septic shock, signs symptoms , syndromes , test, , treatment , trypanosomiases, type 1 polyglabndular syndrome, adrenalities, acute granulomatous, autoimmune, Addison disease, idiopathic, adrenal morophology , cytokines , cytomegalovirus, , tuberculosis, adrenal morphology , Adrenal mass(es). Adrenal adenorna(s); Adrenal carcinoma; Adrenal neoplasm(s) incidental. Adrenal neoplasm(s), incidental Adrenal medulla, activation , catecholaminergic, blood supply , definitive zone , diffuse neuroendocrine system, disorders . niso Pheochromocytorna cerebral effects , dental aspects , diagnosis , imaging , infancy childhood, MEN2, respiratory effects , embryology , endogenous opioids , fetal, fetal zone , ganglion cells , histologv , normal, histopathology , hormones produced by, hyperfunction , hyperplasia , children, pheochromocytorna, hypofunction , inhibitory dynamic tests , paraganglioma , hvperadrenocorticism (Gushing disease. Gushing syndrome), response autonomic nerve impulses, somatostatin , stress response, substance P , Adrenal medullary insufficiency, infancy childhood. Adrenal neoplasm(s), Adrenal adenoma(s); Adrenal carcinoma amenorrhea, benign malignant, biochemical differentiation , sonographic differentiation , cortisol-producing, Gushing disease/syndrome, differential diagnosis feminizing, glucocorticoid-producing, Crooke cells, hyperandrogenism, incidental. causes , clinical management , differential diagnosis , fine needle aspiration biopsy functional capacity , determination , incidence , surgical management , localization , plasma ACTH levels with, genital hormone producing benigh versus malignant, size clinical significance , virilizing, Adrenal nodule(s), Adrenal neoplasm (s) autonomous, bilateral, imaging , black (pigmented), cushing svndrome, as incidentalomas, yellow, Adrenal steroid inhibitors, Adrenal tumor(s). Adrenal neoplasm(s) Adrenal vein, left, management , adrcnalectormy, right, Adernal venous sampling, hyperaldosteronism, with pheochromocytoma, technique , Adrenarche, cause , definition , exaggerated, physiology , premature, Adrenergix pathway, benigh proastatic hyperplasia plasia , Adrenergic receptor(s), affinities catecholaminergic agents,
critical illness, downregulation , upregulation , alpha-Adrenergic receptor(s) stimulation , intracellular mechanisms activated by, Adrenergic receptor (s) stimulation , intracellular mechanisms activated by, hepatic, stimulation , intracellular mechanisms activated by, desensitization , glucose metabolism, renal, skiin, stimulation , intracellular mechanisms activated by, thyroid hormones , alpha-Adrenergic receptor agonist(s) calcitonin secretion, epinephrine as, norepinephrine as, PTH secretion, renal response , p-AdrenerSic receptor ae;onist(s) calcitonin secretion, epinephrine as, hCG synthesis, leptin secretion, PTH secretion, renal response , Adrenergic receptor antagonist(s) effects endocrine function values, hyperkaleinia caused by, lipid levels, preoperative use , children, Adrenergic receptor antagonist(s) alcohol , type 2 diabetes treatment, calcitonin secretion, diabetogenic properties , effects endocrine function values, hypoaldosteronism caused by, lipid levels, marfan syndrome, overdose, glocagon therapy , postpartum thyroiditis, PTH secretion, theraphy with, cardiac manifestations hyperthyroidism, glaucoma, with diabetes, Craves disease, adverse effects side effects , hyperthyroidism, pregnancy, pheochromocytoma, pulmonary edema, postpartum thyroiditis, preoperative, with pheochromocytoma, children, silent thyroiditis, subacute thyroiditis, P-Adrenergic transmitter(s), protein metabolism, Adrenocortical carcinoma, aldosterone overproduction , clinical features , diagnosis , pathophysiology , treatment , Adrenocortical insufficiency. Hypoadrenocorticism , infancy childhood, primary, secondary, ti-ratnirnt , SIO primary, clinical characteristics , diagnosis , etiology , renal effects , secondary, infancy childhood, clinical characteristics , diagnosis , etiology , Adrenocortical rests accessory, intraovarian, intratesticular, tissue distribution , Adrenocortical tumor(s), hypoglycemia, Adrenocorticotropic hormone (corticotropin, ACTH), Corticotropin, exogenous actions , fetal life, adrenal androgen synthesis, adrenal embryology, adrenarche, age-related changes , alcohol intake , aldosterone secretion, with aldosterone-producing adenoma, dexamethasone-suppressible hyperaldosteronism, amenorrhea, bioassays , cell source , central nervous system effects , cocaine use , cortisol secretion, Gushing disease , deficiency , a!so Adrenal insufficiency, adrenal insufficiency caused by, adrenal morphology inb, clinical manifestations , congenital, etiology , growth hormone deficiency , infancy childhood, infants children treatment , isolated, laboratory assessment , maintenance therapy , pituitary or hypothalamic causes , differential diagnosis , postoperative, clinical management , radiation-related, sings symptoms , transient functional, treatment , effects endocrine function values, elderly, embrvo, eosinopenia, excess. Hyperadrenocorticism clinical manifestations , fetal, fetal lung maturation, functions , glucocorticoid excess, glucocorticoid secretion, cytokines , half-life , human chorionic, hypersecretion . Hyperadrenocorticism hypersensitivity , hypothalamic pituitary-adrenal systerm, immune response, immunoinhibitory properties , lordosis, female rats, lung cancer, lymphocytes, measurement , melanotropic action , minernlocorticoid excess, ocular effects , pancreatic islet cells, paraneoplastic (ectopic) secretion , Ectopic ACTH syndrome adrenal cortex , bacterial infections , elderly, hypertension , infection risk, laboratory findings with, localization lesion , pregnancy, renal effects , parturition,periodic hormonogenesis , pituitary adenoma producing. Pituitary adenoma(s), corticotrope plasma/serum level anorexia nervosa, clinical applications , critical illness, acute versus chronic phase, Gushing syndrome, depression, endothelin , , measurement , indications , medullary thyroid cancer, pregnancy, renal failure, precursor , plasma/serum level , pulmonary disorders, production by lymphocytes, HIV-infected (AIDS patients, placental, pulmonary, actions ,radioirnmunoassays , reference values , resistance, conhgenital, familial, riboflavin ,secretation age-related pattersn , corticotropin-releasing hormone , depression, critical illness, cytokines , diurnal pattern , dynamic pattern , dynamic testinc , feedback regulations , by gastrointestinal tumors, glucocorticoids , natriuretic peptides , by pheochromocytoma, regulation , mechanism , response hemorrhage, rhythms , somatostatin , by somatostatinoma, stress~,, substance P , thymus , skiin, steroidogenesis, stress response, synthesis , therapy with glucocorticoid therapy, comparison , indications , tobacco use , tumors producing, Adrenocorticotropic hormone, paraneoplastic (ectopic) secretion ; Pituitary adenoma(s), corticotrope characteristics , MENI, treatment , two-site immunoradiometric assay , vitamin C , Adrenocorticotropic hormone (corticotropin, ACTH) infusion test drug effects , expected response , intraindividual variation , method , physiologic factors affecting, special considerations/interpretation , substance measured , Ldrenocorticotropic hormone (corticotropin, ACTH) receptor(s) defect, isolated, gene , G, , intracellular signaling pathways , lymphocytes, mutations , related endocrine disorders, Adrenocorticotropic hormone (corticotropin, ACTH) test, assessment HPA function glucocorticoid-treated patients, interpretation , low-dose short, short, critical illness, interpretation , depression, , Adrenocorticotropin. Adrenocorticotropic hormone (corticotropin, ACTH) drenodoxin, Adrenodoxin reductase, Adrenogenital syndrome, Congenital adrenal hyperplasia definition , dental/orofacial abnormalities , plasma ACTH levels , Adrenoleukodvstrophv, adrenal insufficiency caused by, neonatal, neuropathy , Adrenomedullin, calcitonin gene family peptides, amino acid sequency, bio-effects , bone metabolism cardiac tissue, central nervous system effects , clinical significance , critical illness, endocrine effects , gene , human, amino acid sequence, immune functions , immunoreactive, plasma/serum level if, adrecocoretical production , renal clearance , secretion , by pheochrornocytoma, tissue distribution , vasodilator effects , Adrenomyeloneuropathy, adrenal insufficiency caused by, Adrenotensin, Adult respiratory distress syndrome. Acute respiratory distress syndrome diabetics, glucocorticoid therapy , inhypercalcemia, leukocyte dvsfunction , plasma /serum levels pulmonary hormoiics, Adult T-cell leukemia/lymphoma, PTHrP expression , Advanced glycation end products (AGEs), formation , diabetic neuropathv, Adynamic bone hypercalcernia with, renal osteodystrophy, Adynamic ileus, with pheochromocytoma, Aerobic energy expenditure, Acrobic exercise, AFCAPS/TexCAPS, Affective illness. Sec Depression phase-advance hypothesis , Afferent pupillary defect, Affinity, hormone-receptor, AFP. Alpha-fetoprotcin, Age, bone age androgen receptor number, brain natriuretic peptide synthesis, breast cancer risk. circadian rhythms, cutaneous biosynthesis vitamin D, diabetic retinopathy, endocrine test results, endometrial cancer risk, endothelin production, fracture risk, glucocorticoid therapv, growth hormone receptor expression growth hormone secretion, insulin resistance, melatonin production, prolactin secretion, TSI-I secretion, type 2 diabetes, AGEs. Advanced glycation end products, Aggarwal, Bharat B., Aggressiveness, with androgen replacement therapy, Aging, effects . Elderly adrenal cortex, adrenocorticotropic hormone, aldosterone, anterior pituitary, antidiuretic hormone, autoimmune disease, breast function, calcium regulation, carbohydrate metabolism, catecholamines, diabetes, empty sella syndrome, female reproductive system, gonadotropins, growth hormone, hypercalcemia, hypercalcemia malignancy, hypothyroidism, hypoadrenocorticism, hypothalamus, hypothyroidism, impaired homeostasis, lipid levels. male reproductive system, melatonin, menopause, modular thyroid disease, obesity, osteoprosis, paget disease bone, parathyroid hormone, pineal gland, pituitary adenomas, posterior pituitary, prolactin, renin, thymus, thyroid glands, thyroid-stimulating hormones, vitamin D deficiency, Agonadia, pscudohermaphroditism, Agonadism, true, Agonist(s), partial, Agouti-related protein (AgRP) appetite, central nervous system effects , feeding behavior, leptin , Agouti signaling protein, obesity , Agranular cells Juxtaglomerular (JG) cells;Mesangial cells, extraglomerular Agranulocvtosis, antithvroid drug-induced, 
AgRP. Sec Agouti-related protein AGT. Angiotensinogen Agus, Zaiman S., AI-IC. Adrenal hvpoplasia congenital Ahinia, Rexford S., Ahmann, Andrww J., Ah rcceptor, A-HydroCort, Hydrocortisone sodium succinate, AIDS. AIDS-rclated complex, thyroid function parameters , AIRE (autoimmune regulator) gene, mutations , Air Force Coronary Atherosclerosis Prevention Study/Texas Coronary Atherosclerosis Prevention Study. AFCAPS/ TexCAPS Airway, upper, obstruction growth hormone excess , hypothyroidism, Akt serine kinase, Alanine effects endocrine function values, gluconeogenesis, metabolism', plasma/serum level . Hyperalaninemia; Hypoalaninemia pyruvate kinase, structure , Alanine aminotransferase, plasma/serum level catabolic stress, with defects carnitine metabolism, with fatty acid oxidation defects, P-Alanine transaminase deficiency, Albers-Schonberg disease. Osteopetrosis , Albinism, Chediak-Higashi syndrome, Hermansky-Pudlak syndrome, ocular involvement , oculocutanemous, organ-specific involvement , Abright ereditaryostepdyustrophy,  cutaneous manifestation , genetics , gene mutations , 
Albright hereditary osteodystrophy (continued) normocalcernic variant , TSH resistance , type IA, type IB, type II, Albright syndrome. McCune-Albright syndrome, Albumin, bone, calcium binding , characteristics , glycated (glycosylated), reference values, inflammatory response, inherited conditions affecting, physiologic characteristics , plasma/serum level . Hyperalbuminemia; Hypoalbuminemia calcium balance, critical illness, pregnancy, Sertoli cell secretion , serum levels, disorders affecting, steroid binding by, synthesis , hepatic, thynius, thyroid lioi'nionc binding , Albuminuria, type I diabetes, intensive therapy , Albustix, Albuterol. Sympathormimetics, effects endocrine function values,Alcohol, niso Ethanol abuse. Alcoholism adrenal effects , beneficial effects , blood pressure, breast cancer, cardiovascular effects , common alcoholic beverages, diabetes, diabetic diet, diuretic effect , electrolytes, facial flushing, fuel metabolism, gout, growth hormone, andhyperuricemia, 
hypoglycemia induced by, hypoparathyroidism caused by, ingestion , endocrine brain , intake moderate, by postmenopausal women, estradiol levels, osteoporosis, plasma lipoprotein levels, mechanisms action , mineral metabolism, pituitary effects , pseudo-Cushing syndrome, thyroid effects , vasopressin secretion, withdrawal from, Alcohol flushing, Chlorpropamide induced, Alcoholic ketacidosis, creatinine measurement , ketoaciduria , lactic acidosis metformin , Alcoholism, bone metabolism , calcium metabolism, dupuytren contracture , eating disorder, feminization , growth hormone, gynecomastia , hypophosphatemia ,
hypothalamic-pituitary-adrenal axis , hypothalamic-pituitiiry-gonadal axis ,
hypothalamic-pituitary-thyroid axis , ischemic necrosis caused by, liver disease caused by, magnesium deficiency , osteoporosis , prolactin, serum hormone changes ,susceptibility , CRH hyposecretion, testicular failure, thyroid function , Aldehyde oxidase, molybdenum, Aldesicukin (IL-2), effects endocrine funci values, Aldolase C, Aldose reductase, sorbitol pathway, Aldose reductase inhibitor(s) diabetic cataract, diabetic hand, leukocyte function diabetes, Aldosterone, Mineralocorticoid(s), actions , age related changes blood pressure, acroinegaly, cardiac fibrosis, nnd collagen formation, critical illness, acute versus chronic phase, deficiency , Hypoaldosteronism autoimmune adrenal failure, isolated, primary, secondary, definition , drinking behavior, effects endocrine function values, elderly, glucocorticoid excess, gynecomastia, half-life , hypertension caused by, inhibition by atrial natriuretic peptide, natriuretic peptides , lead excess , mean blood production rate , adrenal contribution , mechanism action , metabolism , hepatic, renal, genital differences , 
sodium intake , metabolites , bioactivity , mineralocorticoid excess. acromegaly, age-related patterns , with aldosterone-producing adenoma, . critical illness, evaluation , indication , , 11beta-hydroxylase disease/17-20-lyase deficiency, infancy childhood, postural stimulation test , pregnancy, renovascular hypertension, plasma sodiul levels, reference values , renal effects , renel tubular unresponsiveness , conditions associated with, resistance, response hemorrhage, salt ingestion, secretion , diabetes, drugs affecting, endothelin , regulation , renal failure, somatostatin , spironolactone , steroidogenesis , pathway , suppressibility , determination , synthesis , inborn errors , target tissues , tobacco use , urinary, age-related patterns , vasopressin secretion, Aldosterone:cortisol (A:C) ratio, blood/plasma
clinical significance , hyperaldosteronism, Aldsterone escape, artrial natriuretic peptide , Aldosterone glucuronide, Aldosterone-18-glucuronide, as index aldosterone production, Aldosterone oxidase. Aldosterone syntha Aldosterone:plasma renin ratio, determinatic , Aldosterone-producing adenorna(s). Adrenal adenoma(s), Aldosterone producting, Aldosterone receptor(s), Aldosterone synthase, activity , characteristics , deficiency , gene , llp-hydroxylasc gene, chimer formed by, reaction catalyzed by, steroidogenesis, Aldosteronism glucocorticoid-suppressible, idiopathic, familial occurrence , 'primary diagnosis , hypertension , laboratory findings , magnesium metabolism , preoperative preparation patient , urinary kallikreins , Alendronate sodium (Fosamax) actions , adverse effects side effects , bone mass, with corticosteroid therapy, dosage administration , effects endocrine function values, esophageal effects , indication , postmenopausal osteoporosis, preparations , therapy with glucocorticoiid-induced osteoporosis, with glucocorticoid therapy, osterporosis, men, prevention, damage administration , Paget disease bone. Alesse, Alfentanil. Narcotic analgesics effects endocrine function values, Aiglucerase, effects endocrine function values, Algodystrophy, diabetes, Alkaline phosphatase activity , as biomarker bone formation, bone, bone metabolism, bone-specific (BAP), as marker bone formation, Paget disease bore, primary hyperparathyroidism, reference values , technical characteristics , deficiency , ocular effects , isoenzymes, reference values , plasma/serum level Graves disease, hepatic origin, hyperthyroidism, hypophosphatasia, as marker osteoblast activity, multiple myeloma, origin , from osteoblasts, osteomalacia, Paget disease bone, renal osteodystrophy, total factors affecting, as marker bone formation, technical characteristics , vitamin D-dependent rickets type 1, primary hyperparathyroidism, reference values , skeletal immunoassays , serum, osteoblast activity, tissue nonspecific, mutations , bone disease hypophosphatasia, Alkali therapy, hypocitricuria, renal tubular acidosis, uric acid stones, Alkalosis. Metabolic alkalosis calcium balance , hypokalemic Gushing syndrome, disorders characterized by, evaluation patient with, llp-hydroxylase deficiency, 17alpha-hydroxylase/17,20-lyaso dcficiencv, hypophosphatemia , pseudoaldosteronism, Alkaptonuria, clinical findings , 2000t cutaneous manifestations , genetics , metabolic defect , ocular effects , organ-specific involvement , urine screening tests , 17alpha Alkylated androgens, delayed puberty, Alkylating agents male infertility, premature ovarian failure, 17alpha-Alkylation, All Acute lymphoblastic leukemia, Allantonic duct, Allele heteroheneity, Allergic reaction(s), androgen replacement therapy, eicosanoids , ocular involvement , skiin, with sulfonulurea therapy, Allergic rhinitis, Addison disease, hypercorttisolism, mast cell , Allergy(ies) endogenous hormones, premenstrual syndrome, IL-3 , IL-4 , IL-5 , insulin. Sec Insulin, allergy Allescherin boydii, thyroid infection, Allgrove syndrome, Allograft rejection, Allopurinol, adenine phosphoribosyltransferase deficiency, effects endocrine function values, management purine overproduction, structure , uric acid stones, Allostasis, Allo-steric regulation, metabolic regulation , Almora. Sec Magnesium gluconate, Alopecia adrenal insufficiency, androgenetic, autoimmune polyglandular (hypofunction) syndrome, diffuse hyperthyroidism, non-scarring, pregnancy, with erythroderma, inherited disorders amino-acid metabolism, females, with glucagonoma, male-pattern, adrenal virilizing syndromes, 5alpha-reductase , type I polyglandular syndrome, Alopecia androgenica, females, Alopecia areata, hyperthyroidism, Alopecia totalis, vitamin D-dependent rickets, Alora. Sec Estrogen(s), transdermcil Alper, Melvin G., Alpha ccll(s). A ccll(s) Alpha-fetoprotein, cmbrvoncil carcinoma , inflammatory response, islet cell tumors , ovarian cancer , as tumor marker ovarian tumors, testiclilar tumors, Alpha-glucosidase inhibitor(s) actions , adverse effects side effects , dosage administration , indications preparations , type 2 diabetes, Alpha-methyidopa. Methyidopa, alpha, Alpha (a) subunit free, measurement , glycoprotein hormones, gene , pituitary adenomas, nho Pituitary adenoina(s), null cell precursor , reference values , testicular function, Alport syndrome, clinical features ocular manifestations , recessive, genetics , X-linked, genetics , Alprazolam, Benzodiazepine(s) premenstrual syndrome,. Alprostadil (Caverjet, Edex, MUSA), actions , adverse effects side effects , dosage administrative , dysfunction, indication , preparations , ALS, Acid-labile subunit, Alseroxylon, rauwolfia derivatives Alstrom syndrome, glucose intolerance , ocular manifestations , otolaryngologic findings , Alternative splicing, Altitude, endocrine test results, Altretamine, effects endocrine function values, Aluminum accumulation, dialysis patients, 
bone disease, effects endocrine function values, intoxication, dialysis patients, with hyperalimentation, ostcomalacia caused by, reference values , Aluminum hydroxide, thyroid function parameters, Aluminum-magnesium hydroxide. Antacid(s) effects endocrine function values, Alveolar hypoventilation diabetics, hypothyroidism, Alveolus/alveoli calcium deposition , hypercalcemia, formation , hypopituitarism, retractile pressure , Aizheimer disease amylin fibrils , estrogen replacement therapy , nerve growth factor therapy , protection against, estrogen , somatostatin , Amadori product, Amalgam tattoo, Amantadine, effects endocrine function values, Amaryl. Glimepiride, Amastia, Amaurotic idiocy, familial. Tay-Sachs disease, Ambiguous genitalia, age at onset , clinical evaluation amenorrhea, congenital adrenal hyperplasia, with CYPl9 (aromatase) gene defects, 17a-hvdroxvlase/17,20-lyase deficiency,inhint with

25 Hydroxy Chol ecalciferol (Vitamin D3), 1,25 Dihydroxy Cholecalciferol (Vitamin D), 17 Hydroxy-progesterone, 17 Ketogenic steroids (Corticosteroids), 17 Ketosteroids, 17 OH Corticosteroids (Ketogenic steroids), 5-HIAA (Hydroxy Indole Acetic Acid), 5 Alpha DHT-Di hydro testosterone, serum, Abnormal Haemoglobin studies, blood, Absolute Eosinophils Count, blood, ACE-Angiotensin Converting Enzyme, serum, Acetaminophen (Paracetamol), Acetone, urine (Qualitative), Acetone,Serum (Quantitative), Acetyl Choline Receptor Antibodies, Acid load test, Acid Phosphatase (total), serum, Acid phosphatase with prostatic fraction, Acidified serum test (Ham’s test)(PNH test), ACTH-Adreno corticotropic hormone, plasma, ACTH stimulation test for DHEAS test, ACTH stimulation test for 17 hydroxy progesterone, ACTH stimulation test for Aldosterone test, ACTH stimulation test for Androsteindione test, ACTH stimulation test for Cortisol test, Acti test, Acti test and Fibro test, Adenosine Deaminase (ADA)

Adenosine deaminase (ADA), Adenovirus, IgM antibody, ADH-Anti diuretic hormone (Vassopressin), Adrenaline (Epinephrine), B- Acid Fast Bacilli,susceptibility-Amikacin, AFB- Acid Fast Bacilli.susceptibility-Augmentin,  Acid Fast Bacilli, susceptibility-Capreomycin, AFB- Acid Fast Bacilli, susceptibility-Cycloserine, AFB- Acid Fast Bacilli,susceptibility-Ethambutol, AFB- Acid Fast Bacilli.susceptibility-Ethionamide, AFB- Acid Fast Bacilli,susceptibility-lsoniazid, AFB- Acid Fast Bacilli,susceptibility-Kanamycin, AFB- Acid Fast Bacilli, susceptibility-Ofloxacin, AFB- Acid Fast Bacilli.susceptibility-Pyrazinamide, AFB- Acid Fast Bacilli,susceptibility-Rifampicin, AFB- Acid Fast Bacilli,susceptibility-Roxithromycin, AFB- Acid Fast Bacilli, susceptibility-Streptomycin, AFB-Acid fast bacilli by Flourescent Microscopy, AFB-Acid fast bacilli by ZN stain, AFB-Acid fast bacilli, Culture (Rapid) by US FDA approved non-radiometric, continuous monitoring VERSATREK System. (Improved version of commonly used manual and automated/radiometric rapid culture systems) AFB-Acid fast bacilli, Susceptibility-1 st line [SIRE] (Includes Streptomycin, Isoniazid, Rifampicin and Ethambutol

Diffrunt Hospital in india are

Moolchand Health Care, St. Stephen’s Hospital, Dr. Ram Manohar Lohia Hospital, G.B Pant Hospital, Lok Nayak Hospital, RG Stone Urological Reasarch Institue, Sir Ganga Ram Hospital, Jeevan Hospital, Deen Dayal Upadhyaya Hospital, Orthonova Hospital, Batra Hospital, Sahi Hospital, Indraprastha Apollo Hospitals, Holy Faimly Hospital, Jeevan Mala Hospital, Jaipur Golden Hospital, Lady Harding Hospital, Smt. Sucheta Kriplani Hospital, Mata Chanan Devi Hospital, Safdarjung Hospital, Mohindra Hospital, M.P Heart centre Hospital, Bara Hospital, Hindu Rao Hospital, Holy Angels Hospital, Shankar Hospital, Holy Family Hospital, Chopra Hospital, Escorts Hospital St. Stephen's Hospital, Vardaan Hospital, Visitech Eye Hospital, Vardhman Mahavir Medical College, Safdarjung Hospital, Lok Nayak Jai Prakash (LNJP) Hospital, Private Hospital, Guru Teg Bahadur Hospital, Kalra Hospital,  Bangalore Baptist Hospital, Bowring Hospital, B W Lions Eye Hospital, Bangalore Hospital, Bangalore Children's Hospital, Genesis Hospital, Lakeside Hospital, M G A Hospital, Mallya Hospital, Manipal Hospital, Sindhi Charitable Hospital, Yellamma Dasappa Hospital,  Sir Harkisandas Narottamdas Hospital, Prince Alikhan Hospital, Saifee Hospital, Breach Candy Hospital, Jaslok Hospital, Habib Ismail Hosptal, Kalajyot Hospital, Motiben Dalvi Hospital, Bombay Hospital, Corwest Jain Clinic Group of Hospitals, D.S. Kothari Hospital, Lotus Hospital, Elizabeth Hospital, Bhatia Hospital, The Bomanji Dinshaw Petit Parsee Hospital, Cumbala Hill Hospital, Noor Hospital, K.B. Haji Bachoali Charitable Ophthalmic & ENT Hospital, Bai Jerbai Wadia Hspital, Nawroji Wadia Maternity Hospital, Wadia Hospital, Yerla Medical Trust Hospital, Tarachand Bappa Hospital, Dhanwantari Hospital, Shri Samasta Parajiya Suvarna Hospital, Smt.B.C.J. General Hospital, Smt.B.C.J. General Hospital, Holi Spirit Hospital, Lilavati Hospital, The Bandra Holy Family Hospital, Raheja Hospital, Gurunanak Memorial Hospital, Ramakrishna Mission Hospital, Radhibai Watumull Global Hospital, BSES Hospital, Sushrut Hospital, LH Hiranandani Hospital, Maganlal Popatlal & Sarvoday Hospital, Rajawadi Municipal General Hospital, Shatabdi Hospital, KBB Hospital, V.N. Hospital, Dr.K.B. Babha Hospital, Cooper Hospital, Bhagwati Hospital, Sion Hospital, K.E.M. Hospital Nayar Hospital, Kama Hospital, G.T. Hospital, J.J. Hospital, Sr.George Hospital, Aliganj Hospital, Blue Cross Hospital, Getwell Hospital, Krishna Lok Hospital, Lucknow Hospital, Medwin Hospital and Health Centre, Vivekanand Hospital, G.M.Associate Hospital, Dufferin Hospital, Civil Hospital, Communicable/Infectious Diseases Hospital Lifeline Hospital and Heart Centre, Lucknow Hospital, Shalimar Hospital, Rajendranagar Hospital Tuberculosis Hospital, Lucknow Rajpoot Hospital, Nishat Hospital, Z A Chartable Hospital, Indira Gandhi Eye Hospital, Sitapur Eye Hospital, Awadh Hospital and Heart Centre, Balrampur Hospital, Cantonment General Hospital, Civil Hospital, Dr.Shyama Prasad Mukherjie Hospital, Fatima Hospital, G.M.Associate Hospital, K.K.Hospital, Veerangana Jhalkari Bai Female Hospital, Shekhar Hospital, Queen Mary Hospital, Osmania General Hospital, Gandhi Hospital, Government Maternity Hospital, Nilofar Hospital, Cancer Hospital, T.B. Hospital, Hospital for Mental Diseases, Sarojini Devi Hospital, Sundram Medical Foundation, Chennai. MIOT Hospital, Chennai. Apollo Hospital, Madras. Malar Hospital Limited, Gandhi Nagar Adyar, Chenni. Calcutta Medical College Hospital. N.R.S. Medical College & Hospital, Calcutta. R.G. Kar Medical College & Hospital, Calcutta. Calcutta National Medical College & Hospital, Instt. of Postgraduate Med. Education & Research, Calcutta Seth S.K.M. Hospital, Calcutta. Sambhu Nath Pandit Hospital, Calcutta. Lady Dufferinvictoria Hospital, Calcutta. Howrah General Hospital, Howrah, Hoogly Sadar Hospital, Hoogly, Chittaranjan Seva Sadan & Chittaranjan Cancer Hospital,  Ramakrishna Mission Seva Pratishthan General Hospital, Calcutta. Bangalore Baptist Hospital, Bowring Hospital, B W Lions Eye Hospital, Bangalore Hospital, Bangalore Children's Hospital, Genesis Hospital, Lakeside Hospital, M G A Hospital, Mallya Hospital, Manipal Hospital, Sindhi Charitable Hospital, Yellamma Dasappa Hospital, 
Aliganj Hospital, Blue Cross Hospital, Getwell Hospital, Krishna Lok Hospital, Lucknow Hospital, Medwin Hospital and Health Centre, Vivekanand Hospital, G.M.Associate Hospital, Dufferin Hospital, Civil Hospital, Communicable/Infectious Diseases Hospital Lifeline Hospital and Heart Centre, Lucknow Hospital, Shalimar Hospital, Rajendranagar Hospital Tuberculosis Hospital, Lucknow Rajpoot Hospital, Nishat Hospital, Z A Chartable Hospital, Indira Gandhi Eye Hospital, Sitapur Eye Hospital, Awadh Hospital and Heart Centre, Balrampur Hospital, Cantonment General Hospital, Civil Hospital, Dr.Shyama Prasad Mukherjie Hospital, Fatima Hospital, G.M.Associate Hospital, K.K.Hospital, Veerangana Jhalkari Bai Female Hospital, Shekhar Hospital, Queen Mary Hospital,

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